Monday 30 March 2009

Smoking & The Eye

The whole lecture centered around the 'brief advice' that us, GPs etc should be giving to smokers. Evidence shows that brief advice given by GPs to all known smokers leads between one and three percent to stop smoking for at least six months. That doesn't sound like a lot but it's above those that would've quit anyway. Anyway doing this brings health gains in the population for a modest spend and will reduce hospital costs long term.

Smoking is still prevalent throughout the uk
  • 26%+ in Scotland
  • 25% in England
  • 23% in Wales
  • 26% in NI
It's 37% in the greater Glasgow area and 50% in some of the more deprived areas. There's 277 hospital beds in Glasgow taken up each day with px suffering from smoking related illnesses. Out of 1000 lifelong smokers, 1 will be murdered, 6 will die in road traffic accidents and 500 will die from a smoking related illness. Here ends the stat parade.

Cigarettes contain 4000 chemicals and 60 of them are known carcinogens. Nicotine is the addictive bit, Carbon Monoxide binds to haemoglobin and causes hypoxia/vascular problems and Tar is the thick sticky ming that causes pulmonary problems. Also you have arsenic, cyanide, ammonia, acetone, formaldehyde etc etc etc! Horrible. Cigarettes aren't allowed to be called light or mild these days because they aren't. Cutting down leads to compensation. So although 'cutting down' seems to do nowt it should still be applauded as the px clearly wants to quit and is making an effort.

How Smoking Affects the Eye
  • AMD - risk is increased 2-3times. Smoking is the only proven cause of AMD that's actually avoidable. Out of 500k people in the uk with AMD 54000 have it because of smoking. This is the kind of info you need to give to px because apparently only 7% of people are aware of the link. Furthermore the yoof of today are more worried about losing their eyesight than they are getting cancer or some ting! 9/10 people said they would quit at the first sign of a problem with eyesight.
  • Cataracts - smoking doubles the risk of nuclear cataract and the risk increases with the amount smoked
  • DR - smoking accelerates development or worsens the existing condition
  • Thyroid Eye Disease - people who have Graves disease and smoke have 4 times the risk of developing eye problems (puffy lids, bulging eyes, squint, gritty feeling, sometimes swelling behind eyeball pressing on ON and disrupting vision). The risk increases with the amount smoked.
  • Optic Neuropathy - caused by reduced blood flow to the eyes. 16x increased risk of developing it w/smoking and developing it at a young age to boot. Like at 51 instead of 64. Not nice
  • Smoking can cause a more serious glaucoma with earlier onset due to again the reduced blood flow to the eyes
  • Children born to smoking mothers are prone to developing squinting, learning disability, smaller size
Other problems
  • Aesthetics - premature ageing, bad breath, smelly, fatter round the middle, crap at sports, yellow fingers
  • Stroke, gum disease/tooth loss, Cancer of lips, tongue, throat, larynx, oesophagus, narrowed arteries, heart attacks, coronary heart disease, chronic obstructive pulmonary disease, bronchitis, emphysema, chest infections, asthma, lung cancer, stomach ulcers, stomach cancer, kidney cancer, bladder cancer, pancreatic cancer, wrinkling of skin, osteoporosis, impotence, reduced fertility, testicular cancer, miscarriage, Sudden Infant Death Syndrome, gangrene, peripheral vascular disease.
  • Quite a few
  • Secondhand smoke also proven carcinogenic in 2002 - ask if anyone in household smokes
Health Effects Again
  • Immediately you have eye irritation, headache, dizziness, sore throat, nausea, cough, reduced coronary blood flow
  • Increases CHD risk 25-35%
  • Increases lung cancer risk 20-30%
  • Risk of acute stroke 82%!!!!!
Benefits of Quitting
  • Start almost straight away - blood pressure goes back to normal after 20 minutes
  • Better wound healing, less likely to have complications during surgery
  • Halts progression of existing conditions eg COPD
  • Mental/financial benefits
  • MED/LONG TERM increases life expectancy if before the onset of serious disease, even if px has serious disease does better job of fighting it
Addiction
  • The compulsive physiological and psychological need for a habit-forming substance
  • Nicotine's actually more addictive than heroin and cocaine and has higher relapse rates
  • It takes 7 seconds to reach the brain. You're looking at about 200 hits per day.
  • In high levels nicotine is a potent nerve poison. It stimulates adrenal glands and thus produces adrenalin which increases heart rate, BP & breathing
  • It also acts on ACh receptors establishing the 'reward pathway'. There's a rush of ACh and new receptors are developed. Dopamine is release and pleasure/enjoyment results
  • Nicotine withdrawal involves urges to smoke, depression, sleep probs, GI upset, headache, restlessness, cravings, increased appetite, poor concentration, irritability & anxiety.
Brief Questioning Tactix
  • Open questions to help them explore the issue
  • How do you think you'd cope w/out a cigarette for a day? What would help you in that situation? What do you like about smoking? What don't you like?
  • Have you ever thought about quitting? Have you tried before, how did that go?
Stuff that works
  • Nicotine replacement therapy
  • Zyban
  • Health professional advice
  • Individual counselling
  • Behavioural support groups
  • Pharmacist
  • No smurking day
  • Workplace tobacco policy
  • Smokeline
Not proven to work
  • Brand switching
  • Cutting down
  • Low tar
  • Alternative therapies
  • That Alan Carr book
In Glasgow
  • Intensive smoking cessation groups citywide as well as one to one brief support at 200+ pharmacies, Breathe (for pregnant women) & Hospital Inpatient Service
  • Intensive support is 1hr/week groups, weeks 1&2 info, week 3 =quit and 4-7 group support. Stops 16-20% of people after a year. Difficult cos it's a chronic relapsing condition. The group is relaxed/informal and can actually be fun
  • Groups don't suit everyone so the pharmacy does trained 1 to 1 counselling ("Starting Fresh") 5-10 minutes once a week and NRT dispensed regularly for up to 12 weeks
  • Groups have shown to be twice as effective as Starting Fresh
  • Heavier smokers find it harder to quit. If smoking within one hour of wakening px needs intensive support.
  • Breathe project launched in May 2004. 4 weeks support and NRT.

Friday 27 March 2009

Nutrition & The Eye

Nutrition is a risk factor in developmental and age-related eye conditions.

Fish Oil
  • Contains the lipid docosahexanoic acid (DHA) which is an essential polyunsaturated fatty acid. It's found in the body as a structural component of membranes (1-4%) and is highly concentrated in nerves and photoreceptors (up to 50%).
  • The DHA increases membrane fluidity (there's a constant need to make new membranes) and improves the efficiency of membrane functions including phototransduction & synaptic transmission
Antioxidants
  • Reminder - oxidation is loss of electrons. When biological molecules are oxidised first an unstable free radical results and then the electron is lost/bond is broken.
  • Free radicals are caused by high energy light and toxins like the ones in pollution, drugs and smoking. The concentration of free radicals in the body is exacerbated by high oxygen levels, poor diet and ageing.
  • Lipids in membranes, proteins (both in enzymes and structural) and DNA (protein synth) are susceptible to oxidation. If the free radicals become too numerous you will get cell damage
  • Antioxidants neutralise free radicals and limit oxidative damage
  • Examples of antioxidants include carotinoids (fruit/veg), Flavinoids (fruit/bilberries/gingko/tea), Vitamin E (seed oils/fruit), Vitamin C (fruit/veg), Zinc & Selenium (meat/fish/nuts/seeds/cereal)
  • Important carotinoids include beta carotene found in carrots and apricots, lutein and zeanthin (found in corn, leafy veg & egg yolk) and lycopine from tomatoes/fruits.
Special Protection for the Macula

Special protection for the macula is useful because the macula is of course always being bombarded by light, has a high metabolic rate (it's constantly transmitting info) and the highest concentration of oxygen in the entire body.

Antioxidants in the macular pigment xanthophyll are lutein and zeanthin and you can get em from corn, cabbage, kale, egg yolk as well as some other fruits and vegetables. Obviously nutrition alone ain't goin' to save ya - MD is multifactorial - genetic risk, race/pigment, vascular health, oxidative damage.

Four age related studies (see P102 shared care book) showed that high doses of an antioxidant zinc substance reduced progress of nuclear cataract and decreased progression of ARMD in those with intermediate or high risk ie those with large drusen and pigment abnormalities.

Nutritional Advice to Px re: eye disease
  • Eat fruit and veg (corn, carrots, spinach)
  • Routine supplements aren't justified if your diet is pretty good already
  • Supplements may benefit those at high risk. If you aren't then they aren't going to do any harm but don't think subbing them for stopping smoking works
  • Other factors like smoking and vascular health may have a greater impact
  • Bear in mind that you aren't a nutritionist - if the px is thinking of taking a load of supplements best to refer them to one
  • Good articles here
Developing Visual System
  • The foetus/neonate needs a lot of DHA for photoreceptors and synaptic membranes and Vit A for photopigments. These are provided by the mother.
  • Preterm infants are especially vulnerable due to low fat levels / limited nutrient stores and receving little or no breast milk
  • Fish? WHO now says 3 servings of oily fish are good every week especially if you're pregnant. Pregnant women transfer the DHA to the baby and can leave their own supply depleted. So if the woman takes supplements then they have a good amount of DHA for both them and the baby. The baby takes only as much as it needs so the supplements don't make much difference to them

Gonioscopy

To view the angle. It's Greek. Specifically it's to view the iridocorneal angle. The primary use of gonioscopy is in glaucoma. There are a few other uses that i'll cover later. Barkan defined the different types of glaucoma with the help of gonioscopy as well as being the first to notice that the superior angle was narrower than the inferior angle. He classified glaucoma into closed and open angle. You can't view the angle with slit lamp alone. The originator of this whole thing was Maximillian Salzmann in 1915. He realised that total internal reflection obscured the anterior angle. He used a Fick type contact lens with radius smaller than that of the cornea. Then in 1919 with the development of the slit lamp a magnified view of the cornea could be obtained and with it a magnified view of the angle. In 1938 after a few less exciting developments that I can't be bothered to cover Goldmann introduced the first indirect gonioscopy mirror lens.

When I say an 'indirect' lens that means a truncated prism which has a surface that basically fits the cornea. Light from the anterior angle comes out, hits a mirror and is reflected into your own human eye via the slit lamp. The difficulty involved in viewing the angle is due to the critical angle of reflection at the cornea/air interface. The lens replaces the eye/air interface and the critical angle is eliminated by the steeply curved outer surface of the lens. Direct gonioscopy (using eg the Koeppe lens) produces an image round to the side of the lens which is much less easy to observe.

Direct Gonioscopy - Koeppe Lens

The Koeppe lens resembles a very thick high plus lens. It only has x1.5 magnification so has to be used in conjunction with another magnifying device like a hand slit lamp. The image produced is erect and virtual

Advantages - panoramic binocular view, good view of narrow angles, transillumination, can be used on bedridden patients, wide fov for teaching purposes

Disadvantages - time consuming, requires large working area as px really needs to be supine. You would have to walk all the way around the patient in order to see the full 360 degree view the lens provides. You might even need an assistant. It needs separate mag and illum, mag is low and it's not great for fine detailed view (eg you aren't going to see schwalbe's line)

Indirect Gonioscopy

Advantages - focal illumination allows the location of schwalbe's line. If you can't see schwalbe's line then you have a very narrow angle. The view is magnified and is excellent for picking out fine detail. It's technically simple to use and useful for lazer treatment.

Disadvantages - stereopsis is difficult, requires coupling fluid which can be a bit messy, observation is reversed and fov is small. An anaesthetic (eg proxymetacaine) is used.

Grading System

There have been many over the years and to analyse them all now would cause me great confusion. So we're going to go with the classic one - Schaffer.
  • Grade 4 = wide open = 40 degrees
  • Grade 3 = open = 30 degrees
  • Grade 2 = just about showing trab meshwork = possible closure = 20 degrees
  • Grade 1 = 10 degrees = eventual closure
  • Grade S = <10>
  • Grade 0 = closed angle
The Schaffer grades correlate well with Van Herick's slit lamp grades for anterior depth. When doing gonioscopy there is a 'goniogram' which is a generic sheet on which you can mark the appearance and grades along with what's visible. This is great to reduce confusion between optom and ophthal or whoever.

Structures

From Cornea
  • Schwalbe's Line
  • Non Pigmented Trab Meshwork
  • Pigmented Trab Meshwork
  • Scleral Spur
  • Ciliary Body
  • Iris Root/Insertion

Optometric Management of Cataract Px

Prevalence
  • Increases with age. Approaches 100% if lens opacities are included
  • Patient defines whether it's visually significant
Risk Factors
  • Exposure to UV light
  • Smoking
  • Heavy drinking
  • Family/Genetic
  • Race (higher in indian population)
Vision in Cataract
  • Decreased light transmission of short wavelength light esp if the cataract is nuclear
  • Refractive change - myopic shift, cyl changes
  • Light scatter - glare, decreased contrast sensitivity, decreased VA
Management Pathway
  • Px attends optom, cataract diagnosed and discussed, risks and benefits of surgery discussed. Px wishes to proceed and info given. Px offered choice of hospital and appointment agreed
  • Px attends HES outpatient appointment w/ophthalmologist, pre-assessment w/nurse, date for surgery arranged/agreed. Details of current medication received from the optom/GP/Px
  • Patient attends HES and day case surgery undertaken
  • Patient attends HES or optom for final check, sight test. Px discharged or 2nd eye discussed and appt arranged

Optometric Management of Low Vision Px

Current Management
  • Optoms are trained and qualified & NHS pays (usually in hospitals only)
  • Low Vision shared care pays for community based Low Vision
Proposed Pathway
  • START: Px referred to LV service from secondary care, GP, social worker, rehab officer, community nurse, occupational therapist, self referral. Px may have RVI, LVI, CVI. All px contacted by LVS within 10 days
  • Px attends LVS - service seamless across health, social care and voluntary sector. Full sight test forms part of assessment. Px given info on eye condition, entitlements etc as well as local services. Counselling and advice on employment/education available. LVAs, advice and home adaptation discussed and made available. Referral to other areas of health and social care as needed. This includes certification.
  • Px has followup visits as needed that may take place in home or elsewhere

Optometric Management of ARMD

Prevalence
  • 350,000-500,000 in the uk 'blind' from ARMD
  • Most of the impairment is due to wet ARMD
  • Most of that is untreatable
Incidence
  • 21,000 new cases every year
  • 10% added risk/year to fellow eye
Non modifiable Risk Factors
  • Age
  • Genetic predisp
  • Gender (females 2xrisk)
  • Race
  • Iris colour (light pig)
  • Type I diabetes
  • Rx
  • Cataract (can vary w/UV exposure)
  • Handgrip strength ('weak' px)
  • OD appearance
  • Size @ birth
Modifiable Risk Factors
  • Smoking
  • Drink
  • Socioeconomic factors
  • Nutrition
  • Body Mass Index
  • Dietary Fat Intake
  • CV disease
  • Hypertension
  • No statin usage (if you have cholesterol more likely to get it)
  • Aspirin
  • Type II diabetes
  • Sunlight exposure
  • Birth of child (px weaked if had 5 or 6 kids)
Optometric Assessment of the ARMD Px
  • History - co-morbidity; hypertension, diabetes, hyper cholesterol, smoking, excessive alcohol
  • Acuity - If poss use logMAR - more immediate steps so can spot disease early
  • Contrast sensitivity
  • Amsler - History of metamorphopsia, give copy to put on fridge
  • Refraction
  • Dilated assessment w/Volk lens
  • Decisions - if WET refer. Will see vasc changes, exudate, haem, oedema, neovasc
  • Decisions - if DRY monitor especially if they have large confluent drusen
Medical Management of the ARMD Px
  • Flu Ang exam to clarify/make diag of wet/dry
  • Treat: Laser Photocoagulation - leakage away from the fovea
  • Treat: Anti VEGF treatment - intraocular injection which blocks neovasc and causes shrinkage of existing vessels
  • Macular translocation - surgery for ppl w/huge scars
  • Future: retinal implants. Currently only for ppl who are totally blind

Diabetic Retinopathy

Background
  • Microaneurysms
  • Retinal haemorrhages +/- any exudate
Pre-Prolif
  • Venous Beading
  • Venous looping
  • IRMA
  • Multiple deep round haemorrhages
  • Cotton Wool Spots
Prolif
  • NVD
  • NV Elsewhere
  • Pre-retinal/vit haem
  • Pre-retinal fibrosis/tractional ret det
Photocoagulation
  • Focal/grid macular
  • Peripheral scatter
  • Pain
  • No driving that day
  • Loss of visual field
Maculopathy
  • Exudate within a disc area centred on fovea
  • Circinate/group of exudates within macula
  • Retinal thickening within a DA centred on fovea (seen on stereo photo)
  • Microaneurysm/haem within a DA centred on fovea if assoc with best VA of 6/12

Thursday 26 March 2009

Optometric Management of DR

History
  • before insulin DR wasn't recognised and insulin dependent diabetics died early. Type II were diet controlled
  • 1950s-1970s diabetes becomes a controlled disease and DR was recognised. Hypoxia was recognised as the trigger for the neovascular process
  • Laser treatment - pan retinal photocoagulation - focal treatment to stop specific leaks
  • Newest development c.2000 - intensive diabetic control via continuous control, slow release insulin, insulin pumps
There were two big trials
  1. Diabetes control and complications trial - type I diabetes (insulin cells destroyed)
  2. UK prospective diabetes study - type II diabetes (high blood glu, relative insulin def)
Glucose tests for diabetes
  • Fasting blood sugar = >7.8mmol/litre
  • Random blood sugar = >11.1mmol/litre
  • Two hour post load = >11.1mmol/litre = you're diabetic
  • Two hour post load = >7.8mmol/litre = you have impaired glucose tolerance
Glycosylated Haemoglobin = Glu bound to haemoglobin. Measuring it is good because it correlates w/blood sugar average over 6-8 weeks:
  • Normal <6% (corr w/ 7.6mmol/l)
  • Controlled diabetic = 9% (corr w/10mmol/l)
  • Intensive control = <7%>
If you're intensively controlled then there's only a 7% chance of you developing DR after nine years and if you're 9% controlled then it's around 20%. So intense control is way beneficial.

Initial Adverse Effects - sudden intensive control leads to increased DR in 6/9 months. Phase it in!

Factors which aggravate DR
  • smoking
  • hypertension
  • hyperlipidaemia
  • obesity
  • renal disease
  • pregnancy
  • puberty
The challenge currently is to get 100% of diabetics an annual eye exam w/high level of accuracy (or every 6 mths for the high risk people) Strategies for screening include
  • GP w/ophthalmoscope - good recruitment but poorest accuracy
  • Special screenings + photo
  • Optom - good accuracy but poorest recruitment. Optom has skill, equipment, accuracy and knowledge
Scottish National Screening Program
  • Want to detect referable sight threatening retinopathy to reduce the risk of sight loss
  • To detect lesser degrees of DR
This was launched April 2002. Target px were everyone >12yrs old, both type I and II, excluding everyone that already has ophth. care, the irreversibly blind and the medically untreatable. Working out national register of all diabetics, system of appts/feedback/followup. The whole thing is audited and coordinated

Screening process
  1. Digital camera - 80% can have non-dilated. Pic stored for analysis
  2. Digital photo w/mydriatics
  3. Volk
Photos are then graded by technicians or optoms

The importance of screening must be stressed to ppl. Remember tell em not to drive after mydriasis/light sensitivity

Results
  • Retinopathy yes/no?
  • Follow-up (screen or referral)
  • GP and ophthalmologist informed

Flu Angiography #2: Abnormalities

Abnormalities can either involve hypofluorescence or hyperfluorescence. Defects are sub-classified according to
  • type of defect (leakage, blockage etc)
  • Location (retina, choroid, optic nerve)
  • depth
  • whether in inner retina (sharp focus) or outer retina/choroid (blurred cos you're looking through retina)
Also timing of defect. Autoflu before injection can occur if there are drusen. Drusen fluoresce a bit even without NaF. Sclera and gliomas can too. Then you have early arterial filling defects, later arterio-venous leaks and late phase abnormal staining and leaks (lack of staining indicates non-perfusion).

HYPOFLU - FILLING DEFECTS
  • Indicate not enough circ. Occlusion or insufficiency of artery/vein
  • @Retina - central or branch occlusion of art/vein OR hypoflu showing optic nerve atrophy
  • @ON - normally there's a bright flush @ arterial phase and venous phase & late staining. In cases of ON atrophy you get hypoflu and only late staining present. This could happen in glaucoma
  • Choroidal
HYPOFLU - BLOCKED TRANSMISSION
  • Retinal vessels not visible eg pre-retinal haemorrhage - blockage in media or anterior retina
  • Choroidal vessels not visible indicates deep retinal/subret haemorrhage
HYPERFLU - ABNORMAL VESSELS
  • Dilated and or tortuous, wide & bright
  • Neovascular vessels - wide, leaky, high density
  • Tumour Vessels
  • Aneurysms/leaks
HYPERFLU - LEAKS
  • Microaneurysms - initially contained, then hazy leaks. EG Background DR
  • Inflammation causing leakage like ischaemic optic neuropathy
  • Choroidal Pooling - breaks in the RPE and NaF pooling in subret space eg central serous retinop.
HYPERFLU - PRE-INJECTION FLU
  • Autoflu - drusen
  • Pseudoflu - not really fluorescing, just bright reflective surfaces - hard exudates, coloboma
HYPERFLU - TRANSMITTED FLU
  • lack of pigment, retinal atrophy
  • choroidal background brighter 'window defect'

Flu Angiography #1

Assessment of retinal circulation is important in oc. disease. Loss of circ is the main cause of visual loss in the western world - diabetic retinopathy, ARMD etc. The flu angio reveals vascular dysfunction that you can't see and make the extent/severity more visible

NaF is non-toxic and maintains good flu. in biological tissues. 60% of it binds to protein in blood and 40% is in solution. The flu absorbs short wavel. light (blue) and emits longer (yellow/green)

Retinal flu
  • Central Retinal Vessels - tight junctions in the epithelium, no NaF leaves the vessels normally
  • Choriocapillaris - fenestrated epithelium. The NaF in solution passes straight out into the extracellular space and the NaF that's bound to the proteins will stay inside the capillaries
  • Ciliary Body - Unbound NaF can leave vessels, join the aq. and colour the anterior chamber
  • Optic Nerve - served by central retinal circulation (no leakage) but leakage in the choroid nearby will stain glial tissue and this is late staining which occurs after a few circulations of flu through the bloodstream
  • Sclera - collagen is stained by flu from the choroid
Methods of Angiography
  • Injection - IV, rapid concentrated inj. Arm --> Heart --> Lungs --> L Heart --> Arteries --> CRA (takes 7 secs to get there)
  • Observation w/fundus camera fitted with cobalt blue filter
  • Risks/side fx - staining of skin and urine, 5-10% of px nausea and vomiting, rarely serious allergic reaction
Normal Flu Angiogram
  • Arterial Phase - 7-9 secs after injection. This is the arm --> retina circulation time. The stained blood appears to be flowing up the middle of the arteries ("lamellar flow"). The choroid fills in a patchy way giving background flu.
  • Venous Phase - 10-12secs --> 15secs. Arteries contain NaF, the capillaries are filled and the veins also begin to flow. The flu flows along the sides of the veins. Bright background from the choroid, bright filled arteries, veins w/lamellar filling, maximal brightness @ 14-18secs
  • Late Phase - diffuse NaF in the blood, veins and arteries equal in brightness, background will be diffusely stained, staining of the optic nerve and sclera. This is 4-5 minutes after injection.

Glaucoma Mega Brief Version

Progressive optic neuropathy associated with elevated IOP. Has characteristic field defects - nasal step, arc scotoma etc. Lowering IOP is protective even in normal tension glau.

Fluid in the eye is produced and drained away at a certain rate. Glaucoma doesn't involve a problem with increased production, it's a drainage issue. You can help by lowering the production though. Normal IOP is considered to be 21 or less. Ocular hypertension is when pressure is higher than normal but the px doesn't have glaucoma. There's a diurnal fluctuation - usually higher in the AM.

Goldmann/Perkins is used if optom is unsure whether to refer or not. History can give clues - hyperopes are more prone to closed angle glaucoma whereas myopes are more prone to open angle. If the patient has had refractive surgery then the change in rigidity/flatness of the cornea could lead to underestimation of the pressure. Also side effects of asthma treatment could increase chance of glau.

Damage may progress as follows - if some nerves are damaged then the surrounding nerves can follow. It's thought that using some drugs the surrounding nerves can be spared. Research into this is in progress the noo.

The optic disc is imaged mostly with the direct ophthalmoscope but indirect biomicroscopy is becoming more widespread in practice. Also fundus photography, stereo imaging and computerised topography. Disc assessment concerns the cup and disc size. The disc should follow the ISNT rule, if it doesn't then suspect glaucoma. The NRR should be thickest at the inferior, then superior, then nasal, then temporal. Also look for focal loss of ON fibre tissue and disc haemorrhages.

Assessment of the anterior chamber angle can be with Van H's on the slit lamp, ultrasound, Gonioscopy - shaffer grades.

There are various types of glaucoma - open and closed/narrow angle, chronic and acute, primary and secondary, congenital, juvenile and adult.

With pseudoexfoliative glaucoma small flecks of white material are noticed in the pupil (on the edge of the lens). They are very easily seen if the pupil is dilated. Other types include pigmentary, uveitic, steroid induced and rubeotic.

Angle Closure glaucoma assessment. As I typed before closure is more likely in a hypermetropic patient. Ask if any headaches/intermittent blurred vision, haloes around lights (corneal oedema), whether taking anti-deps, drugs for asthma etc. If px has IOP of 60 or something could do gonioscopy, instil pilo, acetazolamide, peripheral iridotomy, surgery (trabeculectomy/lens extraction)

Angle closure typically involves small eyes, a mid-dilated fixed pupil, iris atrophy, ON cupping/field loss

Normal Tension Glaucoma - 10-30% of glaucomas are actually normal IOP. This is mostly older PX. If you can lower pressure more still by using medication or surgery then you can slow down progression.

Pilocarpine causes constriction of ciliary muscle fibres, opens trabecular meshwork, increases outflow, causes miosis, headache and low vision in dim light, also contributes to myopia, cataract and accommodative spasm. Instilled 4x/day

Adrenergic type drugs like epinephrine increase aqueous drainage. They can cause adrenochrome deposits (black granules in conj), mydriasis (angle closure glau), rebound hyperaemia and allergic reaction

Alpha-2 agonists like apraclonidine are not often used. They decrease production and increase drainage but cause dry mouth, allergy, headache and fatigue.

Beta Blockers - beta receps in eye moderate aq production so blockers decrease secretion. They become less effective over time and can cause bronchospasm which in asthma px could actually cause respiratory failure and even death if given enough drops. Preps inculde timolol, betaxolol

Prostaglandins increase outflow via trabecular and uveoscleral routes. eg latanoprost. 50% of px are on this. Can cause increased iris pigmentation, thicker eyelashes and hyperaemia. It's expensive too.

Carbonic Anhydrase Inhibitors like acetazolamide are used in the short term for px w/uveitis. Side effects include nausea, lethargy, metallic taste, kidney stones, all. reaction. Combos of the above are also used.

Surgery could involve iridotomy, iridectomy, laser trabeculoplasty. Probs include plain old failure of it to work, accelerated cataract, endophthalmitis

Shared Care: Optometric Management of Glaucoma

The glaucoma prevalence for europeans - one percent of ages 40-60, 2% of over 60s and >3% of those over 80. The risk factors are as follows
  • Age, gender (F), race (african/afro-carribean)
  • IOP, ON Head, Myopia/Hypermetropia
  • Diabetes, systemic hypertension
  • Family History (FH)
  • Smoking, alcohol, socio-economic factors
If IOP is less than 15 glau is very unlikely. Anything between 15 and 25 is low tension. 25 upwards the risk of POAG goes up from 10% to about 35%

People with high diurnal variation are more likely to have glaucoma also. A glaucoma patient also has poor VA in mid range contrast sensitivity. You wouldn't normally spot this as most optoms just use a normal snellen w/high contrast. You could spot it w/a pelli robson though. Blue/Yellow fields are more sensitive when spotting the early visual loss.

There is ganglion cell damage in glaucoma. There are two theories behind this
  1. Magno vulnerability where selective loss of magno cells (concerned w/good flicker/motion perception and more sensitive to low contrast stuff
  2. Redundancy whereby magno and parvo cells are lost at the same rate but the larger number of parvo cells means the effect is less pronounced at high contrast
Glaucoma Management Pathway
  1. Patient attends optom, has sight test. IOP = >21 w/applanation tonometry and/or vfd and/or suspicious discs. This results in the patient/optom making an appointment with an optom with a specialist interest in glaucoma or an OMP
  2. Px attends that person and a full assessment is carried out according to protocol. A decision is taken whether the patient has ocular hypertension (OSI/OMP reviews) or can be discharged (return to optom) OR has glaucoma (treat or refer to HES). The px is advised and given further info
  3. OSI/OMP relays data to HES and the HES reviews the data, advises regarding management and sets up review at HES if needed
  4. OSI/OMP manages px in community setting w/regular reviews set in place. OSI/OMP relay data to the hospital if there's significant progression for HES review if req
Each different shared care scheme has its own recruitment, testing and re-referral criteria. Varies. eg Bristol - people who are included have stable POAG, pigmentary or pseudoexfoliative glaucoma but other glaucomas (ie stuff that needs surgery) is going to be excluded.

Referrals in Optometry

The quality of yr referral letters is a major determinant of your REP as well as being crucial for effective px care and to your reputation in the community.

As well as the referral letter other stuff that comes under referral includes verbal and written communications with the patient and other health care profs allowing transfer of care based on your examination. The referral letters need to be complete, accurate and useful (clear concise and understood)

Info for the Px (verbal or written - both if poss)
  • Reason for referral - diag, risk - "the pressure in your eye is a bit high so you might have glaucoma". Use technical term and description
  • Expected Management - when, who, what
  • Appropriate time course - when expected/followup, what to do if followup is missed
Info for the professional
  • Who is the patient - complete identification and contact deets - phone no. important if it's urgent
  • Reason for ref - heading Re: , the key points, complete and concise relevant supporting info. brief history and test results if relevant. GOS18
  • Identify Yourself - Name, posn, tel no, supervisor if pre-reg, stamp (esp if locum)
  • What you expect/plan to do - recall/monitor/await discharge from hopital
  • Authorisation - was verbal, signature best
Types of ref. out
  • direct referral / shared care scheme
  • ref to consultant via GP
  • ref to optoms who specialize (keratoconus)
Ref Stats - How Well We Are Doin'
  • 2.5% optoms no name on letter, 6% no postcode
  • eg glaucoma - 85% give disc app, iop, field plot. which is good
  • 80% good on the routine info - VA, refraction, symptoms, poor info on previous VA, fundus info, media, onset and duration of symptoms
  • 57% no legible name, no practice address 7%
Types of referral "in"
  • Individual professionals - optoms, GPs, Other health care providers - health visiting nurses, physios, occupational therapists
  • Screening programs - schools, special needs
  • Informal - patient referrals (cos of your good rep - build the good rep and acknowledge this stuff!)

Formal Co-Management Schemes

Currently in the uk there are around
  • >65 diabetic schemes
  • >35 cataract
  • >20 glaucoma/ocular hypertension
  • 13 low vision
  • 10 referral/prioritisation
There are also some broad schemes like the one on the south side of Glasgow.

The Crown Report of 1999 recommended optoms as independent prescribers. The govt decided that supplemental prescriber status would come first. The New Prescribing Advisory Commitees drafted the legislation for that stuff in 2000, including a drug list, specific training and associated ongoing CET.

The New(est) Optom Legislation
  • In 2005 the prescribage became no longer confined to 'in an emergency'. Also the drug list was updated
  • Section 60 Order: student registration is now required, new registration for misconduct, new requirement for malpractice insurance, CL supplies regulated, optometric specialities defined, CET requirement
  • Supplementary Prescriber - this involves a voluntary association with an independent prescriber (w/medical qual) managing cases according to set management plans
  • Independent Prescriber - establishes diagnosis, directs clinical management and is responsible for prescribing
The NHS goals for co-management are as follows:
  • Shorter waiting lists, greater px accessibility and more efficient use of the hospital eye services and the consultants
The benefits to optometry are thisssss
  • Px continuity and advancement of the profession mainly - more variety, interest
  • Increased REP
  • Income to a certain (small) extent

Co Management / Shared Care Intro

Optoms should have an in-depth knowledge of diseases that are the focus of current co-management schemes, the necessary skills required for the schemes and understanding of their current state in UK optometry

There was a change in how shared care related to optometrists in the year 2000. Before then it only involved the optician spotting an injury/disease of the eye and referring the person to a medical practitioner. In 2000 the GOC put forward the following:

If in the pro. judgement of a registered optician there is no justification to refer a person consulting him to a registered medical practitioner the registered optician may at his discretion decide not to refer that person but in that event:
  • He shall record a sufficient description of the injury or disease in his records
  • his reason for not referring
  • details of advice tendered
  • if appropriate and with the consent of the px inform the GP
Legal posn - The NHS (General ophthalmic services amended 1986)

Optom having accepted persuant to the regs an application for the testing of sight make such examination of the patient's eyes as may be required and in doing so exercise proper care and attention. Where a contractor is of the opinion that a patient whose sight he has tested
  • Shows on examination signs of injury or disease in an eye or in its immediate vicinity or any other abnormality of the eye or the rest of the visual system which may require medical treatment
  • If px not likely to attain a satisfactory standard of vision notwithstanding the application of corrective lenses he shall so inform the px's doctor
Optoms have always been involved in px management, they are actually often the initial point of contact for a patient in the community. There are four levels of management which the optom is involved in
  1. Detection with non-specific referral of the abnormality. This is like basic entry level!
  2. Detection with informed referral for advice/management - this is where you're indicating diag/diff diag with maybe some signs and symptoms, level of urgency, suggest what will be done/who will do it. Ideally this is what you're aiming for. Doing (1) is just weak mang. Acceptable, but weak.
  3. Detection w/a directed management plan in which you (a) advise the GP of the status of an ocular condition that you plan to monitor - diagnosis/plan or (b) advise the GP of an ocular condition and provide prioritized referral advice - w/diagnosis/plan, referral pathway and urgency and a note of where you participate in the scheme (maybe later on - like post cataract check)
  4. Formal Shared Care Scheme - this is set up thru the health board/NHS and involves specific training, criteria for px inclusion, examination requirements and criteria for referral and monitoring. This can include delegation of medical management to the optometrist

Sunday 22 March 2009

LV: Types of Telescope

Basic types
  • Hand held
  • Clip on
  • Spectacle mounted
  • Monocular versus binoc
  • Focus/Afocal
Specials
  • Autofocus
  • CL telescope
  • IOL lens scopes
  • BTLT - Behind the lens telescopes. These offer cosmetic adv but great care must be taken obvs. Increased risk of eye injury if the patient bumps into something while wearing them. Also v expensive
  • Bioptic - for px who fulfil certain requirements driving with bioptics is legal in some US states, not here. Px can resolve fine detail like road signs
Contact Lens Telescopes
  • These use a galilean system where the obj lens is a high plus spec lens and the eyepiece is a high powered -ve CL. The vertex dist of specs is equal to the sum of the focal lengths (length of the telescope)
  • The FOV is better than with a conventional telescope cos the exit pupil is really close to the eye but it also depends on the objective diameter (use a blended aspheric to avoid a ring scotoma)
  • Has many practical and cosmetic disadvantages though so is rarely used.
  • EG a +20DS spec lens and a -40DS contact lens. Px is emmetropic.
The fitting of
  • check that normal scope improves VA as expected
  • Check nystagmat for oscillopsia
  • Fit CL, maximise vd of trial frame, perform over-refraction
  • CL must be stable
  • Vergence amplification means separate reading specs are required
  • Account for uncorrected ametropia - +10D hypermetrope with -30CL effective eyepiece is -40 and -10 myope with -30 CL effective eyepiece is -20.
Advantages of
  • In theory px could drive under uk law if visual requirements were met
  • Can get acuity much better than expected in congenital nystagmus
  • IOL could be used instead of CL giving a longer vd so better magnification but IOL can't be changed if acuity worsens
Disadvantages of
  • Low magnification so only works for moderate acuity loss
  • Patient has to be adapted to CL wear & it's difficult to insert contact lenses
  • Need to wear the system regularly for long periods
  • Adaptation to spatial distortion required
  • Poor cosmesis with high vd and big fat plus lenses

Telescopes: Depth of Field

If you do calculations it appears that plus lenses and telescopes have the same depth of field, but px feel it to be less in telescopes. This could be because the dept of focus for the telescope represents a smaller portion of the working space. Anyway here are some more lovely calculations

Ok, the px has 0.5D depth of focus. What change in incident vergence or object distance could the px tolerate:

a. With a plus lens mag of 10D

f = 10cm if obj at focal point

let emergent vergence = +0.5D
L' = L+F = 0.5-10 = -9.5cm

let emergence vergence = -0.5D
L' = L+F = -0.5-10 = -10.5cm

So overall there's a 1cm range

b. With a 2.5x scope with a +4.00

Emergent vergence Ltel = L'tel/M2 = +/-0.5/2.52 = +/- 0.08m

The incident vergence at the reading cap is of course -4D to match up with it so

delta l = (1 / -4D+0.08D) - (1 / -4D + 0.08D) = (1 / -3.92) - (1 / -4.08) = 1cm

NB Aberrations may also have an effect on the depth of field.

NB2 Changing the obj posn can alter the vergence of light entering the eye. This could create a refractive correction for spherical ametropia
  • Move obj closer to magnifier: light divergent leaving magnifier so uncorrected myopes need to hold material closer
  • V.V. ie uncorrected hyperopes need to hold material further away
  • The effect is similar for both plus lenses and telescopes but because of vergence amplification the effects will be much more severe for telescopes

LV: Compensating Telescopes for Ametropia

Again there are three methods for this

1. Full correction for refractive error behind eyepiece

This is the simplest method in which the telescope is clipped onto or held over the spec correction. You could also fit a correcting lens into a holder behind the eyepiece if required. This method doesn't have any effect on the magnification of the system because the telescope is still afocal (unmodified).

2. Partial correction for Rx over objective lens

This achieves some divergence or convergence of light entering the telescope which is then amplified by the telescope to give the correct amount. This is complex to work out and rarely attempted.

3. HERE WE GO MORE F'IN CALCULATIONS: Changing the separation of Fe and Fo
  • Shorten the scope to correct myopia and lengthen for hypermetropia - how much depends on Rx
  • Changing the length has an effect on magnification
Astro Example

Work out the magnification and length of a 3x astro (Fe +60, Fo +20) used by a -10.00 myope with the correction behind the eyepiece. NB With a myope you want divergent light coming out of the eyepiece which can then bend into the patient's eye thru the specs

a. The magnification is as for an emmetropic user as the telescope is afocal so = -Fe/Fo = -3x
b. t = fo' + fe' = 50mm + 16.7mm = 66.7mm

Ok that was easy, but what about if the guy was uncorrected and the telescope was focussed to compensate?

t = fo' + fe' = 1/20 + 1/60-(-10) = 64mm

it's like a part of the Fe power has been borrowed to correct the ametropia leaving it as +70D
So to correct for myopia you need to decrease the sep between the lenses w/Astro
And to correct for hyperopia you need to increase the sep between the lenses w/Astro

the Fe in that case will go down to +50D and the length will increase to 70mm.

Galilean Example

3x Fe = -60, Fo = +20 fixed for -10 myope

t = fe' + fo' = 1/20 + 1/(-60+10) = 0.05 - 0.02 = 0.03m = 30mm
Mtel = Fe / Fo = 50/20 = -2.5x

Conclusion

Myopia
  • Higher mag with focussing astro
  • Increase length of astro telescope to correct
  • Higher mag with correction behind eyepiece for Galilean
  • Decrease length of Gal to correct
Hypermetropia
  • Higher mag with corr.behind eyepiece with astro
  • Decrease length of astro to correct
  • Higher mag with focussing Gal
  • Increase length of Gal to correct
These egs used large ametropias so effect less pronounced usually. Any big cyls >2.00 will have to be corrected behind the eyepiece. Removing spec correction = shorter v.d. and wider field of view but if the scope is going to be used for spotting then it may not be practical to remove specs. Telescopes are mainly used for near vision due to the increase in working space which is usually needed for manipulative tasks like writing or sewing or some ting. The problem is the fov is far smaller so this counteracts that whole thing a bit.

Saturday 21 March 2009

LV: Focal Telescopes

These telescopes can be modified for different object distances and to correct ametropia. Both Gal and Astro.

Vergence Amplification (Freid's Formula)

Vergence entering telescope L = 1 / l

Vergence leaving L' = M2L / 1-t M L where M is the magnification and t the tube length

For practical purposes t is very small compared to l so we can simplify to L' = M2L

eg How much accomm required when a patient uses a 3x telescope for reading at 25cm?

L' = M2L = 3 squared x 4 = 36D - would have to accomm 36D which is too much even for kids

Adapting telescopes for near and intermediate viewing
  1. full correction for viewing distance over objective
  2. increased correction for viewing distance over eyepiece
  3. increasing separation of Fe and Fo
1 Full correction for viewing distance over obj
  • 'Reading cap' over objective lens. Telescope + reading cap = telemicroscope
  • Used particularly w/Gal system
  • Frc has focal length equal to the required WD
  • Parallel light enters telescope and parallel light comes out
Total mag of telemicroscope Mtot = Mtel x Mrc (Mrc = Frc/4)

eg What magnification is achieved when using a telemicroscope w/ 3x telescope and a +4D reading cap?
= 3 x 4/4 = 3x magnification

working space frc = 1/4D = 25cm

So I can see why they use em - to get 3x with a plus lens the working dist would be tiny. Like 8.33 cm. But here it's 25cm which is way more tolerable. Increased working space makes binocular viewing more practical too - that's only possible with about 2.5x with plus lenses, but 5x w/telescopes. You do need to angle the tubes properly though. NB the overall working distance is increased by the length of the telescope too.

2. Increased correction for viewing distance over eyepiece

This isn't a practical method unless the patient already has high-powered reading add for another purpose. Magnification is difficult to calculate.

3. Increasing the separation of Fe and Fo

  • A practical method but more difficult in spec mounting - becomes more front heavy as the tube length increases. Difficult to angle if binocular
  • More common in astros cos they are hand held usually anyway and the image quality is better maintained
Example

2.5 Gal (Fe = -50D, Fo +20D) focussed for 25cm using a 4D reading cap

Mtel = -Fe/Fo = -(-50)/20 = 2.5x
Frc = 1/l = 1/0.25 = +4.00
Mtotal = Mtel x Mrc = 2.5 x 4/4 = 2.5x

t = f'e + f'o = -0.05 + 0.02 = 30mm

Example 2

2.5x gal focussed for 25cm by changing length so reading cap is 'incorporated' in Objective lens

so t = fo' + fe' = 1/Fo + 1/Fe = 1 /20-(1/0.25) + 1/50 = 0.0625 + (-0.02) = 42.5mm

Mtel = -Fe/Fo = -(-50)/16 = - (-50)/20-(1/0.25) = 3.13x

Frc = 1/working space = 1/0.25 = +4.00

In conclusion higher mag can be achieved by focussing the telescope rather than adding a separate reading cap BUT this causes a large increase in length. Not always practical cos the lens housing cannot expand by this amount and it may make the mounting unstable

Example w/Astro

What are the mag and length of a 3x astro telescope Fe = +60, Fo = +20 focussed for 12.5cm w/reading cap

Mtel = -Fe/Fo = -60/20 = -3x
Frc = 1/working space = 1/0.125 = +8.00D
Mtotal = Mrc x Mtel = 3x2 = 6x
t = fo' + fe' = 1/20 + 1/60 = 66.7mm

Example 2

3x astro focussed for 12.5cm by changing length

Mtel = -Fe/Fo = 60/20-8m = -5x

Frc = 1/0.125 = +8.00D

Mtot = Mtel x Mrc = -5 x (8/4) = 10x
t = fo'+fe' = 1/Fo' + 1/Fe' = (1/20-8) + (1/-60m) = 83.3mm + 16.7 = 100mm

Conclusion

For both astro and gal focusing will always provide higher magnification than a reading cap because Mtel increases when Fo decreases, but there are severe practical limitations re increase in telescope length. NB closest focus distance = longest position of telescope (t=max)

LV: Telescopes!

These are a method of optical magnification which is more versatile than the plus lens.
  • Distance, intermediate or near
  • Hand held for spotting or spec mounted but rarely worn constantly/whilst mobile
General formula (for afocal telescopes) is M = -Fe / Fo and also t (tube thickness) = fe' + fo'

Two types of telescope - astronomical and galilean

Astronomical
  • Both eyepiece and obj lens are +ve, this gives -ve magnification and an inverted image
  • Both focal lengths positive so the telescope is long
  • Prism erecting system needed too
  • Heavier, longer and more expensive than galilean
  • Larger field of view and better image quality though
  • Exit pupil outside system
  • Mag range from low - high. Up to 10x
Galilean
  • Eyepiece lens is negative and objective lens positive. +ve mag so erect image
  • One focal length is negative so the system is shorter
  • Lighter, shorter and cheaper than astronomical
  • Poorer image quality and small field of view though
  • Low magnification only - 4x maximum
  • Preferred for spec mounted due to lower weight and shorter length
Labelling
  • Magnification 2.2x, 3x etc
  • Weight in grams
  • Visual field 250m/1000m linear OR eg 12.5 degrees (200 degrees without scope)
  • Working distance 45-200cm
  • Sometimes written 8 x 40 where first number is mag and second number is diameter of objective lens in mm
Exit Pupil
  • This is the image of the objective lens seen through the eyepiece. All of the rays entering the objective lens pass through the exit pupil.
  • The exit pupil defines the field and the amount of illumination. These can be determined by direct measurement or calculation. Ideally the patient's pupil should be exactly the same size
  • Astros have EP behind Fe so patient's eye can get close to it, with Gal the exit pupil is within the telescope so there's a greater distance between it and the eye
diam of exit pupil = diam of obj lens / magnification of telescope

eg size and location of the exit pupil for a 3x24 gal telescope with an Fo of +10.00D

Fe = -3x10.00 = -30.00

1 / l' = 1 / l + F = 1 / -0.0667 + (-30) = -45

1 / -45 = -22.22cm SO EXIT PUPIL IS 22.22cm IN FRONT OF THE EYEPIECE

24/3 = 8mm SO EXIT PUPIL IS 8mm IN DIAMETER

Factors Affecting Field of View
  • 1/Mag - use minimum power - use min until px becomes practiced
  • 1/vertex dist - 5mm = smallest dist w/o specs, 20mm = w/specs & eyecup
  • Object distance
  • Object diameter - but telescope becomes heavier and more difficult to handle
Matching the size of the exit pupil to the px's pupil optimises the field BUT if alignment slips even a tiny bit part of the field will go dark, so in practice it's actually better if the exit pupil is greater than the patient's pupil despite some loss of field as a result - the misalignment is more easily tolerable. Better to measure in practice than calculate as more closely related to px experience.

LV: Flat Field/Bar Magnifiers

Flat Field Magnifiers

  • Self Illuminating - if the light path of obj to eye is reversed can see that all incoming light is focused at the plane of the magnifier
  • Height of a flat field magnifier can be less than, equal to or greater than r (radius)
Magnification of Flat Bar Magnifiers

M = nr / nr + l (1-n)


for a hemi-spherical magnifier where thickness = radius of curv

t = l = r

The thicker a flat field magnifier is in relation to r the greater the magnification. The image is formed very close to the object so there is no relative distance magnification. Patient must wear near correction. Also you can have bar magnifier with magnification in one direction only - plus cyl lens

Advantages
  • Normal reading posture
  • Very bright - gathers light
  • Good for px w/hand tremor
  • Clear image across the lens w/minimal aberrations
  • Combine w/spec mounted to get good mag while maintaining fairly distant viewing
Disadvantages
  • Large lenses - heavy
  • Magnification typically low
  • Reading Material has to be flat and on firm surface

LV: Stand Magnifiers

The stand magnifier allows the correct lens - object distance hands free. However they are more difficult to illuminate and can restrict access to the working plane (eg when writing). There are both fixed focus and variable focus types.

Optical Sitch

Stand height is usually less than the focal length and the object not at the focal plane but within the focal length. This means
  • the light is divergent when it leaves the magnifier
  • the image is a finite distance from the eye SO the eye must have appropriate accommodation or near VA to view the image at this apparent distance - add or accomm must neutralise the emergent divergence
  • the amount of acc/add needed depends on the mag to eye distance - more accom/add needed as they get closer together
  • ALSO assume the vertex dist for the add is zero for stand mag calcs.
Example Calculation

Stand mag of +20DS has height of 4.5cm. What near add would a totally presbyopic px need if the mag to eye distance was 10cm and the eye was very close to the magnifier?

F = 20D
l = 0.045m
z = 0

l' = 1 / l + F
l' = 1/0.045 + 20
l' = -42.22222cm

so...1/-42.222222cm = 2.36D of accommodation req so +2.25 add

if eye 10cm from magnifier then l' = -42.22222 + (-10) = -52cm so add = 1/0.052 = 1.91D so +2.00 add req

Calculating Magnification

+20D magnifier 20cm from reading add, stand height 4cm

L1 = 1/l1 = 1/-0.04 = -25D
L1' = -25D + 20D = -5D
this means light from object is diverging 5D when leaving magnifier

L2 = 1 / (1/L1') - z
L2 = 1/ (1/-5) - 0.2
L2 = -2.50D

Feq = FM + FA -zFMFA
Feq = 20 + 2.50 - (0.2 x 20 x 2.50)
Feq = +12.50D

so M = 12.5/4 = 3.125x

If you hold the magnifier 5cm from the reading add instead

L1 = 25D, L1' = -5D
L2 = 1/ (1/-5) -0.05
L2 = -4D

Feq = FM + FA -zFMFA
Feq = 20 +4 - (0.05x20x4)
Feq = 20D

so M = 20/4 = 5x mag

SOOOOOOO The higher the near add and the closer the eye to mag distance the higher the magnification.

Determining Emergent Vergence in Practice w/out Calculation
  • View Object Thru Magnifier and find max plus lens which still gives clear view when held up against it
  • Use a telescope to eliminate effect of accommodation
  • As the first one there but reverse the light path and focus a distant object on the object plane of the magnifier eg a ceiling light imgd on page and again use plus lenses
  • NB Often companies give max allowable near add which = add when eye->mag dist = zero. Add decreases as eye moves away from it
Advanatages of Stands
  • Good for px with hand tremors
  • Avail w/ or w/out illumination
  • Portable
  • Relatively inexpensive
  • Some patients find it easier to use than hand mag
Disadvantages
  • Limited FOV
  • Accomm or bif required
  • Difficult to determine equiv power
  • Can be bulky
  • Posture/fatigue probs
  • Illumination difficult if it doesn't have it built in

LV: Prescribing +Lens Magnifiers

Plus lens magnifiers are either hand-held, spectacle mounted or stand mounted.

  • Hand-helds can be held at any dist from the eye
  • Most common, often bought by patient
  • Low Powered <16d fov =" 40mm">
  • Med Power = 16-32D 4x-8x spherical up to 25mm size, 50mm size if aspheric, FOV = 10mm at 25cm from eye, 150mm with magnifier close to eye, likely to have internal illumination
  • High Power = >8x, doublet/triplet lens up to 20mm lens diam, 25mm view with magnifier close to eye, have 'folding' design
Hand Held Advantages
  • Familiar to all px, cheap, easy to prescribe
  • Good to carry around for short duration survival reading
  • Can have long eye to magnifier distance (although this gives poor FOV)
  • Could help for px w/reduced peripheral fields
  • Most are compact, lightweight and portable
  • Can have internal illumination
  • Equivalent power varies depending on how it's used
Hand Held Disadvantages
  • Can be ineffective if used with reading add
  • Difficult to maintain correct position for long periods
  • Requires steady hand eye coord
  • FOV is poor w/long eye to magnifier distance and more distortion as lens held further from eye
H-H Instructions
  • Lay magnifier on page and pull away til clearest image
  • Use distance Rx or add depending on preferred magnifier-eye distance
  • Get biggest FOV by holding magnifier as close to eye as possible
  • Most curved surface towards patient's eye for least distortion
  • Hold parallel to reading material and move head and magnifier and heed as a unit
Spec Mounted

Type
  • Edged lenses to fashion frame
  • Paired lenses w/base in prism in standard frame
  • Lenses glazed to special mounting or carrier
  • Clip on
  • Other LVA for occupational use
Binoc If Possible
  • larger FOV, greater depth of field, better acuity, depth perception
  • More conventional appearance, psychological preference/advantage
  • Can provide w/clip on occluder if they want
Contraind.
  • No evidence of binocularity at appropriate distance - use worth 4-dot, mallett, bar reading
  • Don't use if more than 2x difference in acuity
  • No good if central distortion in a previously dominant 'worse' eye
  • If binoc VA worse than monoc
  • If there's too great a convergence demand and the px experiences diplopia or discomfort
Calculating Near Centration
  • NCD = PD x WD / WD + a
  • a = vertex distance PLUS half axial length of eye (eg axial length 24mm = 12mm)
  • eg axial length 24, PD 65, vert dist = 12mm, working dist 10cm
  • NCD = 65 x 100 / 100 + 24 = 52.41mm
  • SO lenses need to be decentered by a much larger amount and this must be done when testing patient
  • Even if eyes look thru optical centre the convergence requirement is considerable eg 1D base out for each mm of PD to view at 10cm
  • Therefore base in prism may be required
Adv of Spec Mounted
  • Hands free, widest FOV due to short eye to mag distance
  • Similar appearance to normal specs
  • Useful for prolonged reading
  • Can incorporate astigmatic correction
  • Good if px has hand tremors
Disad
  • Close working distance, illumination could be difficult
  • Limited magnification if binocular
  • Blurred vision if looking up into distance altho bifs and half eyes are available
  • inc power = inc working distance which could lead to fatigues, headaches and dizziness
  • Reading speed is reduced
  • Add-wise, +6 = easy, +8 = tricky, +10 tricky, +12.00 highly unlikely

Wednesday 18 March 2009

LV: Equivalent Power Of Hand Held Magnifiers

Feq = F1 + F2 - dF1F2

If a hand held magnifier is used in combination with a near Rx or bifocal it's essentially a lens system, so you can calculate equivalent power.

Example

Px has +2.50add and 10DS magnifier 25cm from their bifocal

Feq = 10 + 2.5 - (0.25 x 10 x 2.5) = 6.25

so Meq = 6.25/4 = 1.56x

Rools
  • If magnifier used w/near add and magnifier to eye dist = less than magnifiers focal length the equivalent mag will be higher
  • if greater than focal length then mag will be lower
  • if equal to the focal length then the mag will be equal to the magnifier's nominal mag

LV: Field of View of + Lens Magnifiers

There is an equation which I can't be arsed to derive. It's for linear field of view.

y = D/aFm

y = linear fov
D = diameter of magnifier
a = lens to magnifier distance
Fm = power of magnifier

NBZ
  • the actual field size is less than calculated due to aberrations
  • magnifier must be held as close to the eye as possible if you want to maximise the field of view BUT lens to eye distance has no impact on retinal image size
  • Give the minimum magnification required - don't prescribe for deterioration
Example Calculation

"How does the field of view change when a patient holds a 90mm round +6.00D magnifier 25cm from his eye and then brings it up to a distance of 6cm from his eye?"

D = 90mm, Fm = +6.00D a=0.25m or 0.06m

so from 25cm away y = 0.09/(0.25 x 6) = 60mm
and from 6cm away y = 0.09/(0.06 x 6) = 250mm

Rules to live by
  1. Magnifier held at 1 focal length from eye ->> FOV = diameter of the lens
  2. Magnifier held at 1/2 focal length from eye ->> FOV doubled
  3. Magnifier held at twice focal length from eye ->> FOV 1/2 diameter of lens
  4. etc etc etc etc etc etc
NB AGAIN - Remember to take into account acuity reserve when prescribing - px needs to be a little bit above the threshold to read fluently - ie when reading a book. If just using for 'spotting' in supermarket or something this isn't as important.

LV: Depth of Field of + Lens Magnifiers

When we talk about depth of field we want to calculate how much the reading material can be moved before a patient notices defocus. Here's an example:

Depth of focus = 0.8D (this means 0.4D each side of the focal point)
Simple magnifier of +5.00DS is used

Method 1

the equation we use is

Δl = (1/L2 - F) - (1/L1-F)

we do one of these for each direction you can move in, and then add them together

NB L'2 = -0.4D if obj moves towards lens and +0.4D if the object moves away from the lens
so here we go

Δl = (1/5.4) - (1/-5) = 0.0148m = 15mm
Δl = (1/4.6) - (1/-5) = -0.0173m = 17mm

so total depth of field is 15 + 17 = 32mm

Method 2

Δl = -ΔL'/(4M)squared

Δl = 0.8/(4 x 1.25) squared

Remember to square both numbers of the denominator

Δl = 0.8/25 = 0.032 = 32mm

The conclusion here is that moving the object by only 16mm causes a significant amount of blur (0.4DS here) which will be perceived by the patient. Note that this example involves low magnification. The higher the magnification, the smaller the depth of field will be.

Monday 16 March 2009

LV: Plus Lens Magnifiers 1

M = F/4 as long as the object is at the focal point. So a +8.00D lens has 2xMagnification at 12.5cm.

If the patient's object is at the focal point of the lens then he/she mustn't wear a near add. NB If a patient doesn't seem to be achieving the expected magnification their current specs may not have a +4.00D reading add and the baseline acuity may be incorrect. Always measure vision at 25cm with a +4.00D add to determine a correct baseline acuity.

For younger patients who can accommodate adequately the magnification =
Visual angle subtended by magnified image/angle subtended by object viewed at 0.25m

If the object is at F then the image the patient looks at will be at infinity.
  • Object at F, L=F and L'=0
  • Image is at infinity so no near add is required
  • The Eye to magnifier distance has no effect on magnification
If the object isn't at F there are two possible situations that could arise

1. General Case

M= qL/1-dL'

where q = 0.25m assumed near point
d = lens to eye distance
L = incident vergence
L' = emergent vergence

M depends both on the distance of the lens from the eye and the distance of the object from the lens.

2. Image at near point

M = 1 + F/4

The object is between F and the Lens. The image is produced at 0.25m ie the near point so a +4.00D add or accommodation is required. The magnifier is held close to the Px's eye (d=0). This is known as maximum magnification/manufacturer's magnification

LV: Magnification

Magnification is "after" retinal image size/"before" retinal image size. It is needed in LV when the rx doesn't create enough improvement for the patient to do visual tasks. It can be achieved by
  1. Increasing the size of the object M=new obj size/old obj size
  2. Decreasing viewing distance M=old viewing distance/new viewing distance
  3. Real image/transverse magnification (eg CCTV) M=size of image/size of orig.obj
  4. Telescopic magnification = angle subtended by telescopic image (theta')/angle subtended by original object (theta)
Different methods of magnification can be combined - in this case the total magnification is found by multiplication. For example - "What is the mag obtained by changing print size from 6mm to 15mm, and visual distance from 30cm to 10cm?"

M1 = new/old = 15/6 = 2.5x mag
M2 = old dist/new dist = 30/10 = 3x mag
Total = 2.5x3 = 7.5x mag

Increasing Size Of Object
  • Large screen TV
  • Large print stuff - books, typewriter, telephone, bank statements
  • Photographic enlargement/photocopying
  • Computers - large monitors/keyboards/enlarged screen display
Changing Viewing Distance
  • Simple and effective for distance or near vision - sitting closer to TV
  • eg moving from 12ft to 6ft away from TV = 2x magnification
  • Bringing print from 30cm away to 5cm away = 6x magnification
Real Img/Transverse Magnification
  • Mainly electronic magnification systems
  • Mainly NV but also systems for distance and intermediate tasks (eg video systems with autofocus)
Telescopic Magnification
  • Versatile optical method for D, I & N

LV: Quick Guide to Prescribing LVAs

DISTANCE (Snellen or Decimal)

BVA = starting point and TA = target acuity

M=BVA/TA

eg Px needs to see 6/12 but can only see 6/60
M = 60/12 = 5x mag required

Available LVAs
  • Telescopes
  • Monoc
  • Binoc
  • Fixed focus
  • Variable focus
  • Autofocus
NEAR

eg Px can see N24 at 25cm with +4.00D add and needs to see N12
M=24/12 so 2x magnification needed

Choice of magnifier
  • Keep it as simple as poss
  • Start with lower mag if poss as it allows a bigger FOV and a longer working distance which makes it easier to use
  • Consider illumination
  • Consider the task, the age of the patient (also physical infirmities) and the cost
Magnifiers available
  • Brightfield/Paperweight
  • Illuminated stand
  • Stand
  • Illuminated hand
  • Hand
  • Spec mounted - binocular or monocular

LV: NV Charts

TNR Near Chart
  • N point notation - 1 point is 1/72 of an inch
  • Easy to convert N12 = half the size of N24
  • Rule - divide DV by 3 to get VA@0.25m eg 6/60 = N20 @25cm
Bailey-Lovie
  • logMAR chart w/2 to 6 words on a line depending on word length
  • Words are unrelated to minimise guessing. Not a good test of overall reading
  • Difficult for people w/poor cognition/english
Keeler A
  • Log scale so 1.25x intervals from A1 to A20. A7 = newsprint
  • A1 = 5 mins of arc at 25cm
IOO Chart
  • Rapid assessment of acuity
  • Reading text using words kids can recognise in a random order
  • Approximate log scale
PNAC
  • Designed to establish px's NVA rapidly, ranges from N80 to N5 but uses N and log notation.
  • Equal no of words and letters on each page
  • Words are related to each other so better for young kids
  • Paragraphs of common print size are on the back
Sloan M system (USA)
  • 'M' notation @ 40cm eg 1M = letters subtend 5 minutes of arc at 1m, 2M...2m etc
  • 1M = newsprint
MNREAD
  • Continuous text reading-acuity charts
  • Measures speed and accuracy as well as print size
  • Can also establish size of print that gives highest reading speed

Tuesday 10 March 2009

Varilux Lenses

Liberty
  • Soft design
  • Cheapest - not much of a jump up from bifs
Comfort
  • Wider field without head movements
  • Soft periphery
  • Lens design changes w/increasing add power
Comfort Advans
  • Has rear surface design so comfort design is maintained whatever the prescription (high cyl)
Ellipse
  • Very wide distance vision area (140 degrees)
  • Short progression length
  • Good for small frames
Ellipse Advans
  • Digitally surfaced for 145 degrees DV
  • Near zone aligned so that it matches wearer's natural convergence
Physio
  • Prettay good
  • Lens reduces Coma so increased sharpness in DV
  • Bigger area of clear DV compared to latest varis
  • Greater visual and postural comfort
  • Fitting height 17mm
Physio Short
  • Better for smaller frames 155 degrees
  • Minimum height 14mm
Physio f-360 degrees
  • Personalised version for better near and intermediate (increased intermed width)
Ipseo
  • bestest
  • Personalised depending on wearer's head/eye movement ratio

CL Aftercare

Pre-Advice
  • Come in wearing lenses
  • Daily wear px come in PM so effects of daily lens wear can be seen, extended wear AM so effects of overnight wear can be seen
  • Bring in solutions/case
Initial Discussion
  • RFV, any problems? comfort & vision ok?
  • Recent lens wearing history - max wearing time, how many days/week, how many hours in today?
  • Probs handling lenses? Solution/care system ok?
  • Any other eye problems?
Vision w/CLs
  • Snellen/logMAR acuity monoc/binoc
  • Ret w/trial frame, assess quality of reflex (this may indicate lens lifting off central or peripheral cornea
  • Duochrome useful
Assessment of Fit (Soft)
  • Comfort
  • Vision
  • Corneal Coverage
  • Centration
  • Movement on blink
  • Lag on upgaze
  • Push-up
  • K Mires
  • Any conj compression/buckling of lens edge
Assessment of Fit (RGP)
  • White light - lens position wrt lids, primary & tertiary gaze, lens centration
  • Blue light - flu patterns
Also look for
  • lens/eye interactions eg lens edge near 3&9 o'clock staining
  • Blinking pattern
  • Head position (may indicate loose lens)
  • Eye movements
  • Palp Ap size - can reduce w/RGP, increase w/soft lens
Keratometry over lens
  • Can assess fit, front surface wettability
Inspect Lenses
  • Knick/tears
  • Edge or body defects
  • Deposits - protein, mucus, calcium, Fungi, Jelly Bumps, Rust Rings
  • Do wettability/TBUT
Lenses Out

Get px to do it if poss. This allows you to see how they do it, whether they wash hands etc. If it's the first aftercare you should ask them to clean the lenses too to see how they're doing it. Correct any errors. Generally assess compliance.

Slit Lamp

White Light
  • Perioc skin, eyebrows, upper and lower lids and lashes
  • Bulbar conj
  • Tear film
  • Cornea
  • Inf/sup palp conj
Blue w/Flu
  • Corneal/conj staining
  • TBUT
  • Evert eyelids and examine papillae formation on tarsus
Also examine lenses off eye for damage/deposits. Do any necessary supplementary tests. Rose Bengal, ophthalmoscopy if not done for a while etc

Taking Action
  • Same lenses w/different params - shift in Rx/alteration of fit
  • New lenses of same general type - higher Dk/t
  • New lens type - hydrogel to Si-H to improve physiology, RGP to soft to improve comfort
  • Change lens wear modality - extended wear to daily wear
  • Change replacement freq - monthly to daily?
Taking Action: Care Systems
  • Completely change regimen - multipurpose to hydrogen peroxide
  • Change version - 2 step to 1 step
  • Alter protocol - add extra saline rinse?
  • Change to avoid particular preservative
  • Eliminate need for solutions - dailies
Final discussion with patient
  • Reassurance concerning symptoms - eyes often feel dry at end of day
  • Explain why lenses are being changed - more myopic
  • Explain why solns being changed - different preservative to avoid red eyes
  • Answer any other questions raised - px has heard about extended wear
  • Recommended date of next visit - 6 mths?

Contact Lenses: Fit Assessment

RGP

  • Let lens settle for a few minutes
  • Can check centration, TD and movement under white light first - want good centration, staying within limbus, slow and smooth drop on blink, TD should allow ~1mm each side
  • Then check flu pattern w/blue light - areas of dark blue = touch and brighter green = clearance with thicker tear film
Alignment Fit
  • Even thickness of tears
  • Good edge
  • Hint of apical clearance
  • Centres well
  • Drops slowly & smoothly
  • Stays within limbus in all dirs of gaze
Steep Fit
  • Central pooling suggesting BOZR is less than k reading
  • Inadequate edge
  • Centres well
  • Comfort often fine
  • Drops slowly
  • +ve liquid lens
  • Could see bubble if very steep
Flat Fit
  • Central touch and wide edge
  • Discomfort
  • Centres poorly and moves too much
  • Drops quickly and in arc shape on push-up
  • -ve liquid lens
Strategies
  • Lid Attachment - common now that GP lenses are bigger. Has good comfort. The edge of the lens is in contact with the lid during blinking and in the primary position
  • Lid attachment is the natural fit w/minus lenses and gives good tear exchange and comfort. It's more difficult with a plus lens (a -ve carrier is an option). Beware of corneal exposure and moulding
  • Interpalpebral was more common when more people were using the smaller PMMA lenses. The centration is good and there's less flare. This strategy is useful if the patient has an irregular peripheral cornea as it only fits the regular central part.
  • Alignment fit is used for modern lenses - multicurves, aspherics
  • Apical clearance gives better centration if using a small lens.
Overrefraction
  • A steep lens results in a positive tear lens so the minus power of the lens itself needs to increase
  • A flat lens results in a negative tear lens so the plus power of the lens itself needs to increase
Edge Clearance
  • This is the gap between the cornea and the lens edge
  • Poor EC = stagnant tears, binding, staining, discomfort, hard to remove
  • Good EC = easy removal of lens, good tear exchange, improves lens movement

Soft Lenses

  • Can first check comfort and vision - is vision same as best spec Rx?
  • If the lens blurs straight after blinking and then clears = flat fit
  • If vision starts off clear and then blurs this suggests the lens is too steep
  • Can do this subjectively or with keratometer
  • Assess coverage (lens 1mm over each side ideally)
  • Centration - if bit off to one side but all of cornea is still covered = acceptable. If cornea not covered can either fit a larger TD or smaller BOZR
  • Good blink induced movement is 0.2-0.5mm upwards lens recovery straight after blink
  • Excess blink induced movement is >0.5mm upwards recovery following blink - fit larger TD or smaller BOZR
  • Inadequate = <0.2mm>
  • Lag on upgaze - similar params to blink induced movement
  • Push-up Test - lens should move w/slight resistance when pushed through lids
  • If lens is totally free then larger TD or smaller BOZR
  • If movement is sluggish then smaller TD or larger BOZR
Using K Mires
  • STEEP - Mires immediately clear post-blink due to lid compression of lens, then mires blur as lens distorts
  • FLAT - Mires are blurred immediately post-blink due to prismatic effect of lens movement, then mires clear and lens stabilises
Effect of TD/BOZR on Sag
  • Greater BOZR = less sag, flatter fit, looser lens
  • Greater TD = more sag, steeper fit, tighter lens
Misc
  • Thinner lenses move less than thicker lenses
  • Spun cast lenses move less than lathe cut/molded

Monday 9 March 2009

LVA - Acuity Charts

Snellen

Advantages
  • Well known, commonly used standard
  • Very sensitive to blur and ref. error
  • Good size, easy to move closer to px
  • Has O and H for x-cyl
Disadvantages
  • Unequal numbers of letters on each line - crowding not constant
  • No relationship betw line size and number of letters
  • Poor control of contour interaction
  • Designed to measure normal acuity
  • Scale intervals change at non-standard distances
Snellen @ Reduced Distance
  • 6m letter @ 6m has MAR of 1'
  • Px will be able to see a letter half the size when half the distance away
Bailey-Lovie LogMAR Chart

Adv
  • Size/letter spacing equivalent throughout chart (5/row)
  • 1.25x progression (0.1 log) each line
  • All letters equally legible
  • Constant crowding for all VA levels - easy to use at different test distances
  • Final score takes into account all letter that have been read successfully (see later)
Disad
  • not used as routine measure of VA
  • Scoring/conversion not as easy
  • No O for x-cyl
  • Bit big and therefore hard to illuminate
Scoring

  • The size diff between 2 lines on a logMAR chart is 0.1 log unit so 10 to the 0.1 = 1.25 so the size diff is equal to factor 1.25. 3 lines = approx doubling in VA
  • Log score decreases with improving VA
  • Each line = 0.1 log units and each letter is therefore 0.02
  • Doesn't use viewing dist as part of notation. What you do is add 0.3 to the VA score every time the viewing distance is halved.
EG Px reads 0.8 line and 2 letters on line below so logMAR VA = 0.76
then Px reads 0.5 line and 2 letter on line below so logMAR = 0.5-0.04+0.3 = 0.76

  • Reducing viewing dist by 1.25 requires a correction factor of 0.1
  • so 6.0m to 4.8m = 0.1, 6.0m to 3.84m = 0.2 etc
Other charts include sheridan gardener, glasgow acuity and Kay's pics.

LVA "Routine"

Do current DVA/NVA with aid and specs, noting type of chart and distance used, light level, any head turn, record mon/bin VA. Worst eye first is good to keep morale up. Subjective refraction should use large steps/big x-cyl/reduced testing distance if required (px should be able to see 4 lines)

For distance
  • Identify the px's visual task and estimate the required VA for it
  • Measure best corrected VA and estimate mag required
  • Calculate mag
  • Dist generally for younger px, really need to be motivated
  • Often not req as main problem is at near
For near
  • Identify vis.task and estimate VA required
  • Measure BCVA at 25cm with +4.00DS and then estimate mag
  • If central field defect suspected check w/amsler - ecc. fix?
  • Try predicted mag with simple magnifiers - higher add/hand/stand and use good illumination
  • Modify mag if required, prescribe w/full instructions
Other stuff
  • Binocularity - cover test
  • Contrast sensitivity if req
  • Fields, ophth, fundus photo, IOP
Advice/Follow-up
  • Ideally 2-4 wks, may need to change LVAs

LVA Assessment - Intro

LVA is more of a problem solving exercise than the normal exam - not one unique outcome for px (ie 6/6). The main questions to ask is

What does the px want to do and is it going to be possible (expectations could be high or low)
  • Many px may want a better pair of specs but this isn't possible, may be distressed/disillusioned
  • Px must agree with what you want them to do - ie be happy with it
Speed has to be adjusted to fit the patient - an elderly px is going to need more time for subjective for example. Also avoid unnecessary tests. Initially general observation of the px can reveal a lot - bothered by light, physical infirmities, eccentric viewing, guided?

LV-centric equipment includes suitable test charts, real world items, 1.00D cross cyl, large aperture lenses, LVAs.

H&S is very important
  • Pxs wants and needs (may not be the same thing)
  • Questionnaire is useful for learning how they get on with specific tasks (how's watching tv? Do you sit close to it? etc)
  • Specifics qs for distance, near, occupation, mobility, everyday tasks, hobbies, interests, different lighting conditions, onset of impairment, difference between eyes, believed caused, person who referred, ophthal - current and past treatment, when due back there, previous LV assessment, LVAs, live alone, hopes & expectations, registered etc etc etc
Psychological Aspects

Motivation is very important - they ain't going to get anywhere otherwise - the patient must be ready and willing to accept help.

Loss of vision can be assessed using a similar model to the one used w/bereavement
  1. Denial/Shock - can't be happening
  2. Grief - can't do anything now
  3. Anger - it's not fair
  4. Depression/Apathy - why bother
  5. Acceptance - it's going to be ok
Px really needs to be at stage 5 before we can proceed. There's also a self-efficacy model which works best w/gradual vision loss. It's argued that early skill-orientated intervention can prevent loss of competence and foster a sense of personal control essential to successful rehabilitation.

Sunday 8 March 2009

Low Vision - UK Registration Process

Forms
  • Scotland - BP 1, NI - A655, England/Wales - BD 8
  • New forms introduced in England, should spread to all UK soon - LVI, RVI, CVI
The Letter of Visual Impairment (LVI) Form is given to the patient by the optom. This is the self-referral form geared towards the general public. It is supplied by social services and can allow access to social services if required. The patient contacts the council him/herself.

The Referral of VIP (RVI) form is issued by the local ophthalmology department by people like ophthalmic nurses, optoms, DOs, junior docs, orthoptists. Again this applies for access to social services.

The Certificate of VP (CVI) form is completed by a consultant ophthalmologist. It certifies the px as blind and again grants access to social services as well as passes the info on to the census office. NB Access to help is not dependent on registration. The thing is some people don't like to be 'certified blind' because of the social stigma but would still benefit from the help.

The registration process begins with the BP1 or CVI being sent to social services, the GP and the census office for research. Then a social worker goes out to visit the patient to discuss if they wish to be registered and see if they need a community care assessment in which they are assessed to determine their quality of life wrt everyday tasks/items in their house. The patient then can receive the benefits. NB if it's a kid they are registered blind at the age of four unless there's obviously no sight from the beginning of life.

Benefits of being registered
  • Px can access central govt services - need to be registered for these
  • Can also access local authority services, although some are available without registration. The services include the community care assessment, large button telephone, reading light, home help, meals on wheels etc
  • Some services provided by voluntary organisations may be charged for if the patient isn't registered
  • You also get financial help - 50% off your TV license if you're blind (not partially sighted!) and free BT directory enquiries.
  • Transport is free in some areas and you can claim a disabled badge
  • RNIB do talking books among other stuff
  • DLA for people under 65 and attendance allowance for people over 65
Being registered is helpful for people in general - statistics both nationally and for each local area help to determine funding and resource allocation. The estimate is that up to 50% of px who are eligible aren't registered. The main disadvantage is the loss of self-esteem and hope.

The problems with the whole registration thing
  • Optom/Opthal may not even tell px about it
  • Usually done by ophthal as 'last resort'
  • Poor comms betw professionals involved
  • Long waiting times
  • Not enough social workers & they may not be trained in dealing with LV patients